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C251

Recombinant Human HSPb11/PP25

10ug

1200

1080

现货

国产

C251

Recombinant Human HSPb11/PP25

50ug

3520

3168

现货

国产

C251

Recombinant Human HSPb11/PP25

500ug

12320

11088

现货

国产

C251

Recombinant Human HSPb11/PP25

1mg

17600

15840

现货

国产

C251

Recombinant Human PP25/Hspb11

10ug

1200

1080

现货

国产

C251

Recombinant Human PP25/Hspb11

50ug

3520

3168

现货

国产

C251

Recombinant Human PP25/Hspb11

500ug

12320

11088

现货

国产

C251

Recombinant Human PP25/Hspb11

1mg

17600

15840

现货

国产

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  • Catalog# C251
    Source E.coli
    Description Recombinant Human Heat Shock Protein β-11/HSPB11 is produced by our E. coli expression system. The target protein is expressed with sequence (Met1-Ser144) of Human HSPB11 fused with a His tag at the N-terminus.
    Names Heat Shock Protein Beta-11, Hspb11, Placental Protein 25, PP25, HSPB11, C1orf41
    Accession # Q9Y547
    Formulation Supplied as a 0.2 μm filtered solution of 20mM Tris-HCl, 100mM NaCl, 2mM DTT, 10% Glycerol, pH 8.0
    Shipping The product is shipped on dry ice/ice packs.
    Storage Store at < -20°C, stable for 6 months after receipt.
    Please minimize freeze-thaw cycles.
    Purity Greater than 95% as determined by reducing SDS-PAGE.
    Endotoxin Less than 0.1 ng/μg (1 IEU/μg).
    Amino Acid Sequence
    MGSSHHHHHHSSGLVPRGSHMRKIDLCLSSEGSEVILATSSDEKHPPENIIDGNPETFWTTTGMF PQEFIICFHKHVRIERLVIQSYFVQTLKIEKSTSKEPVDFEQWIEKDLVHTEGQLQNEEIVAHDG SATYLRFIIVSAFDHFASVHSVSAEGTVVSNLSS
    Background Heat Shock Protein β-11 (HSPB11) is a stress-responsive protein that is required to deal with proteotoxic stresses. HSPB11 is composed of an IFT complex B composed of IFT88, IFT57, TRAF3IP1, IFT52, IFT27, HSPB11 and IFT20 and is detected in placenta. HSPB11 has beeb shown to form oligomeric complexes and prevent the aggregation of in vitro denaturated aldolase and glyceraldehyde-3-phosphate dehydrogenase in accordance with the chaperone model of HSPB1 and HSPB5. HSPB11 overexpression protected against etoposide-induced cell death that correlated with a decreased release of mitochondrial Cytochrome C into the cytosol. Inhibition of HSP90 function completely abrogated the protective effect of HSPB11. This data suggests that at least in the case of HSPB11, interaction with other chaperone machines besides HSPA1A may contribute to functional specificity and cellular functioning.